Critical Care Medicine: The Essentials by John J. Marini MD, Arthur P. Wheeler MD

By John J. Marini MD, Arthur P. Wheeler MD

Ideally suited to scholars in severe care rotations and citizens, this concise, useful guide offers the necessities of clinical and surgical serious care in an easy-to-read layout. The authors succinctly clarify the pathophysiology underlying scientific problems and the foremost rules of analysis and sufferer administration, emphasizing cost-efficient approaches.

The Fourth version comprises Controversies in severe Care containers in lots of chapters, which in short summarize opposing arguments on debatable issues. different highlights comprise improved dialogue of CT for belly issues, new ACLS instructions, and new fabric on detachable IVC filters, interventional radiologic thoughts for GI bleeding, and use of vascular ultrasound.

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Extra info for Critical Care Medicine: The Essentials

Sample text

Capillary oncotic pressure is reduced by the hypoproteinemia of cirrhosis, malnutrition, nephrosis, or the administration of excessive crystalloid. When the ratio of plasma/interstitial protein concentration falls, pulmonary edema forms at a lower transvascular pressure, especially when the lung is acutely injured. Gross edema formation is unusual when the serum colloid osmotic pressure exceeds Pw by greater than 4 mm Hg, but is increasingly likely at lower values. Although clearly contributory in many settings, reduced plasma oncotic pressure alone rarely explains edema in the face of normal hydrostatic pressures and an intact capillary membrane.

End-expiratory Ppl can exceed its normal value when the expiratory musculature actively contracts, tension pneumothorax is present, or elevated airway pressure at end exhalation increases lung volume (PEEP, auto-PEEP). If PEEP is intentionally applied and exhalation is passive, the relationship between the compliances of the lung (Cl) and chest wall (Ccw) determines the resulting elevation in pleural pressure: ΔPpl = ΔPEEP × [Cl/(Cl + Ccw)]. In the patient with normal lungs and chest wall, end-expiratory Ppl increases by approximately one half of the applied PEEP during passive inflation because Cl and Ccw are similar over the tidal volume range.

However, though not appropriate for patients with irregular rhythms and shock states, monitoring devices that analyze the shape of the arterial pressure waveform do a creditable job during sinus rhythm of tracking and trending the CO within their limitations. Their value is seriously compromised by atrial fibrillation and other chaotic arrhythmias. Simplified Functional Hemodynamic Monitoring Useful classification of hemodynamic compromise can be attempted using a few simple bedside observations that determine mean arterial pressure and index stroke volume.

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