By Johny A. Verschakelen, Walter De Wever
The second one version of Connective Tissue and Its Heritable problems: Molecular, Genetic, and clinical elements is the definitive reference textual content in its box, with over forty% extra pages at the nature, analysis, and therapy of affliction than its predecessor. amassing new examine on problems specific within the first variation in addition to on these formerly excluded, editors Peter Royce and Beat Steinmann give you the most recent scientific and medical details for scientific experts treating affected individuals. gains of this revised and up to date quantity comprise particular experiences of the medical analysis, mode of inheritance, hazard of recurrence, and prenatal prognosis of every inherited connective tissue illness; a radical description of the morphology of connective tissues; a very up-to-date and revised part at the biology of the extracellular matrix; and the addition of syndromes equivalent to craniosyntosis, and problems of sulfate metabolism.
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Extra info for Computed Tomography of the Lung A Pattern Approach
De Wever a Fig. 10. Acute interstitial pneumonia. Ground-glass opacity is associated with linear opacities, creating a crazy-paving pattern b Fig. 9a,b. Usual interstitial pneumonia in a patient with systemic sclerosis. a The dominant pattern is ground-glass opacity, which is located in the dorsal and basal subpleural region of both lungs, suggesting active lung disease. However, at the basal slice (b), there is some irregular interstitial thickening together with some bronchial distortion (arrow), suggesting the presence of ﬁbrosis in that part of the lung Fig.
15). When sharply deﬁned, airspace ﬁ lling by tissue (tumour, granulation tissue) is suggested (Fig. 16), when blurred airspace ﬁ lling by ﬂuid or cells may be the cause (Fig. 15). Similar to ground-glass opacity, to make the differential diagnosis of lung consolidation, it is also very important to know whether the patient’s symptoms are acute, subacute or chronic. Since airspace ﬁ lling is the most frequent cause of lung consolidation, this pattern is often associated with the presence of centrilobular airspace nodules corresponding with early airspace ﬁ lling, as is often seen in diseases that show a bronchial distribution (Fig.
1991; Helbich et al. 1997; Korn et al. 1992). Disseminated ossiﬁcation is a rare condition in which very small deposits of mature bone form within the lung parenchyma and can be associated with chronic heart disease (mitral stenosis), idiopathic pulmonary ﬁbrosis (IPF) or asbestosis (Gevenois et al. 1994). Finally, the drug amiodarone can accumulate in the lung and is in some patients responsible for a pulmonary toxic reaction with interstitial pneumonia and ﬁbrosis. The consolidated lung parenchyma may appear abnormally dense.