A Color Atlas of Comparative Pathology of Pulmonary by Franz Joel Leong, Veronique Dartois, Thomas Dick

By Franz Joel Leong, Veronique Dartois, Thomas Dick

An annual demise toll of two million, coupled with emerging drug resistance, highlights the necessity for the advance of latest medications, greater diagnostics, and a tuberculosis (TB) vaccine. Addressing those key matters, a colour Atlas of Comparative Pathology of Pulmonary Tuberculosis introduces TB histopathology to the non-histopathologists, scholars, scientists, and medical professionals operating, studying, and educating within the box of TB. It includes a hundred colour pictures and illustrations that carry readability to the data awarded. The atlas takes the weird strategy of overlaying a number of species histopathology, arguably the 1st and really in all likelihood the one source to take action. It presents an easy, annotated, and visible presentation of the comparative histopathology of TB in human and animal versions. The editors have compiled details that is helping TB scientists to tell apart among the positive aspects of all significant animal versions on hand and to exploit them with their strengths and boundaries in brain. The publication offers counsel for choosing the simplest animal model(s) to respond to particular questions and to check the efficacy of drug applicants.

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Extra info for A Color Atlas of Comparative Pathology of Pulmonary Tuberculosis

Example text

The expanded air spaces are empty and contain only a few macrophages/histiocytes or desquamated pneumocytes. Under normal conditions, inflammatory infiltrates, especially eosinophils are absent. AH—alveolar histiocyte. centriacinar have been used synonymously with panlobular and centrilobular, respectively (see above). Alveolar Sacs and Alveoli The distal unit of the lung is formed by multifaceted and cup-shaped compartments known as alveoli. Where bronchiolar epithelium is completely replaced by alveolar cells, the air passage is known as the alveolar duct, and this terminates in a semicircular blind end called the alveolar sac, which is surrounded by four or more alveoli.

Normal bronchiolus and adjacent alveoli expressing thyroid transcription factor (TTF-1) in both the bronchial and alveolar cells.

The nature of the largely varying lesion types that MTB generates as its “culture vessels” in humans suggests that the bacillus encounters very different microenvironments in patients. Some are supportive of growth, others not. And indeed, recent work on TB animal models showed that some lesions in some animal models are, for instance, hypoxic, suggesting the bacilli in these lesions cannot grow. Importantly, when nongrowing bacteria in culture are exposed to antibiotics that kill the growing form of the organism, they show “phenotypic” drug resistance (as opposed to genetic drug resistance, due to mutations).

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